Neuroscientists have long sought to explain how recovery from an acute injury can sometimes give rise to chronic pain—and not always in the area of injury. Now, a team at the Department of Neural and Pain Sciences at the University of Maryland, Baltimore, led by Dr. Ronald Dubner, a member of TMJA’s Scientific Advisory Board, has found some intriguing clues to this mystery. They used a rat model of nerve injury, based on constricting one of the branches of the trigeminal nerve that supplies sensation to the face.
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Brain Signals Help the Spread of Persistence of Pain
